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2nd Edition, The Nemechek Protocol for Autism and Developmental Disorders: A How-To Guide For Restoring Neurological Function

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I have noticed that eating something helps it go away almost immediately, so I take all the supplements when I first wake up, followed by a light breakfast, and nausea leaves. I’ve been on the Nemechek Protocol for about a month now. I’m not doing the Rifaximin as of now but that may change. So far, I’m still at the bottom dose of inulin at 1/8 tsp, 1 tsp of fish oil, and 1.5 tablespoons of extra virgin olive oil. I’ve attempted to increase the inulin dosage, but I get severe bloating when I do that. Parents around the world have been using this protocol with great success for their autistic children. For people with celiac disease, gluten — a protein found in wheat, barley, and rye — causes an autoimmune response that damages our small intestine and can cause many other symptoms. The treatment is relatively simple, unless, of course, you’re French and baguette is your comfort food: you just stop eating gluten. There are no pills or transfusions or transplants to fix it — not yet, at least. It’s been an adjustment for me, but I feel better now.

Okay, so that was the easy question, but when it comes to a good autism diet, I don’t know what the right answer is. Celiac disease and autismThe protocol identifies SIBO and brain inflammation as the likely culprits behind developmental delays, ADHD, Autism and more. Well, around day 8 of the Nemechek protocol, I noticed that my lymph nodes weren’t swelling as bad after eating! Granted, it’s only a small difference, but there definitely IS a difference. I can’t tell you how much that means to me to have hope of eating normally again!

In search of the literature regarding regulators of degradation of SHANK3, a paper demonstrating that ERK2 is critical for the marking of SHANK3 for degradation was identified [ 25]. ERK2 phosphorylates SHANK3 to promote its ubiquitination, the “tagging” signal for degradation. The phosphorylation of SHANK3 causes the dissociation of SHANK3 from membranes, where it is degraded and, thus, cannot perform its scaffolding function. Inhibition of the ERK2 pathway increases SHANK3 abundance [ 25], and therefore inhibiting ERK2 has been proposed in the literature as a strategy to help to increase SHANK3 levels [ 25].Well, I meant to update at the 6-month mark, but life has been hectic. So here we are in March 2019, and I’ve been on the Nemechek Protocol for 8 months now. Rather than working directly to increase levels of SHANK3 protein or affect stability, oxytocin is one such molecule that appears to work on downstream pathways such as long-term potentiation [ 35]. Increasing long-term potentiation (LTP) in multiple areas of the brain, including the hippocampus, has been shown to stimulate dendrite maturation and connectivity [ 36]. Decreased dendrite maturation is one of the possible downstream issues in individuals with SHANK3 deficiencies [ 9]. Due to its effects on LTP and its differential expression in the brain, oxytocin was also shown to decrease LTP-mediated pain hypersensitivity, often seen in individuals with ASD [ 36, 37]. This time, I didn’t see the stark, immediate gains I did last time. By day 10 of the meds (last day), my SIBO symptoms had only improved a little. Small Intestine Bacterial Overgrowth (SIBO) occurs when the type of bacteria that’s supposed to live only in your colon get stirred up and migrate into your small intestine. There is a very helpful Facebook group that you can join – https://www.facebook.com/groups/TheAdultNemechekSupportGroup/

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